No matter if you live in an apartment or a house or just would like to ensure your home is free of EMFs, there are a number of ways you can reduce exposure. One of the most effective is to reduce your use of electronic devices. It is also possible to use EMF blocker paint to block EMF radiation from entering your house. Another easy way to protect your house from EMF radiation would be to install an RF shielding canopy. This is a cloth of net that contains EMF shielding. It is utilized to stop EMFs from entering a room. Another option is to have your house equipped with a conductive enclosure. These enclosures are known as Faraday cages.
Numerous studies have demonstrated studies have shown that nonionizing EMF can cause antiproliferative effects in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is thought to involve down-regulation the cancer stem cell. This could explain the long-term responses seen in certain patients suffering from advanced HCC. However, the mechanism behind AM EMF's effects on cancer patients is not yet clear.
Aspects from AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were studied in mice. The tumors were split into 3 groups. First, the group that was unaffected to RF EMF. Another group of participants was subjected RF EMF at frequencies similar to the one used by humans. In the third, they were subjected RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on tumours was assessed against the effect of RCF. emf blockers indicated that tumours treated with HCCMF showed significant shrinkage. However, tumours treated with RCF showed no evidence of shrinkage in the tumour.
The mechanism behind cancer-specific AM RF EMF may be based on the fact that tumor cells require Cav3*2 voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation upon HCC cells is controlled by CACNA1H the protein which mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H could have more broader implications in the treatment and diagnosis of different cancers.
emf blocker of the controls were never exposed EMF from radiofrequency, and fed a standard mouse diet. The tumours in those in the HCCMF group were infected with Huh7 cells at the time they were 5 to 7 weeks old. The tumors were then killed after they had a high burden.
The tumours from the three groups also displayed distinct growth curves. The tumours treated with HCCMF showed a significant decrease in tumour size after 8 weeks. However, the tumors which were treated by RCF didn't show signs of shrinkage. The difference was highly significant. The tumours treated with RCF had necrosis, which is common when tumors are exposed to RCF. The possibility is that this necrosis is caused by an absence of oxygen in larger tumors.
In sum, the results suggest that AM RF EMF has anticancer properties in vitro as well as in the vivo. A number of studies have proven that AM RF EMF produces measurable reduction in tumours in HCC patients. It is possible that AM RF EMF causes these effects because of CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. In addition, AM RF EMF may exert a sustained effect on the development of HCC tumours in the vivo.