No matter if you live in a house or apartment or just want to keep your home free from EMFs There are a variety of ways you can reduce exposure. One of the most effective is to restrict the use of electronic devices. You can also turn to EMF blocker paint to block EMF radiation from entering your home. Another easy way to protect your home against EMF radiation is to install an RF shielding canopy. emf blocker is a fabric made of net which contains EMF shielding and is used to block EMFs from entering a space. Another option is to have your house equipped with an electrical enclosure. https://telegra.ph/EMF-Blocking-and-EMF-Shielding-04-08-10 are known as Faraday cages.
A number of studies have proven that the non-ionizing RF EMF has antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed to be based on the downregulation of cancer stem cells. This could be the reason for the long-term responses seen in some patients with advanced HCC. However, the mechanism of AM EMF's effects on cancer patients is not yet clear.
Aspects from AM RF EMF on HCC tumor growth in vivo were examined in mice. The tumours were divided into 3 groups. One group did not have exposure RF EMF. Second group members were exposed RF EMF at a frequency that is similar to the one used by humans. The third group was exposed to RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on the tumours was evaluated against that of RCF. The results revealed that cancers treated with HCCMF were significantly shrinking. However, tumours treated with RCF didn't show evidence of tumour shrinkage.
block emf behind cancer-specific AM RF EMF could be due to the fact that tumour cells require Cav3*2 voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effects upon HCC cells is controlled by CACNA1H which is a protein which regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H could have more broader implications in the treatment and diagnosis of different cancers.
The tumours in the control group were not exposed to EMF from RF, and fed a normal mouse diet. The tumors in the HCCMF group were treated with Huh7 cells at the time they were 5 to 7 weeks old. The tumors were removed in cases of excessive burden.
The tumours in the three groups also displayed distinct growth curves. The tumors treated with HCCMF saw a significant reduction in size of the tumor after 8 weeks. However, the tumours that were treated using RCF showed no reduction in size. The difference was highly significant. The tumors treated by RCF were able to show necrosis, which is common in tumors that have been that are exposed to RCF. The possibility is that this necrosis was caused by the lack of oxygen in larger tumours.
In conclusion, the findings show the fact that AM EMF has anticancer effects in vitro and in the vivo. Several studies have shown that AM RF EMF produces measurable reduction in tumours for HCC patients. There is a possibility that AM RF EMF produces these effects through CACNA1H which is a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting influence on the growth of HCC tumours in vivo.