Whether you live in an apartment or a house, or you just want to keep your home free of electromagnetic fields, there are a number options to limit your exposure. One of the most effective is to restrict your use of electronic devices. It is also possible to use EMF block paint to stop EMF radiation from reaching your house. Another easy way to protect your home from EMF radiation is to put up a shielding canopy for RF. This is a fabric made of net that has EMF shielding and is used to prevent EMFs from entering a room. block emf is to get your house equipped with an enclosure that is conductive. They are referred to as Faraday cages.
A number of studies have proven how the non-ionizing energy of RF can cause antiproliferative effects in HCC cells. emf blockers behind AM RF EMF's anticancer activity in vitro is thought to be based on the downregulation in cancer-related stem cells. This may account for the long-term responses seen in patients suffering from advanced HCC. However, the mechanism behind AM EMF's effects on patients with cancer is not clear.
Effects from AM RF EMFs on HCC tumour growth in vivo was studied in mice. The tumors were split in three different groups. One group did not have exposure RF EMF. Another group of participants was subjected to RF EMF at the same frequency to that used in humans. In the third, they were subjected to RF EMF at HCC-specific modulation frequencies. The impact of HCCMF on tumors was compared to that of RCF. The results showed that the cancers treated with HCCMF showed significant shrinkage. However, the tumors treated with RCF didn't show evidence of shrinkage of the tumor.
The reason for tumor-specific AM RF EMF may be based on the fact that cancer cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effects upon HCC cells is caused through CACNA1H, a protein that mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H could have wider implications in the diagnosis and treatment of various cancers.
The tumours of the controls were never exposed EMF from radiofrequency, and fed a normal mouse diet. The tumors of the HCCMF group were injected with Huh7 cells when they were 5 to 7 weeks old. The tumors were removed when they showed excessive burden.
The tumours in the three groups also displayed distinct growth curves. The tumours treated with HCCMF had a significant reduction in tumour size after 8 weeks. However, tumors treated with RCF did not show any shrinkage. The difference was substantial. The tumors treated by RCF had necrosis, which is common when tumors are that are exposed to RCF. It is possible that this necrosis is caused by the lack of oxygen in larger cancers.
In summary, the results indicate the fact that AM EMF has anticancer activity in vitro and in vivo. emf blocker have demonstrated that AM RF EMF produces measurable reduction in tumours within HCC patients. It is possible that AM RF EMF causes these effects because of CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting influence on the development of HCC tumours in living tissue.