Whether you live in an apartment or a house, or you just want to keep your home free of EMFs, there are a number of ways you can reduce exposure. One of the easiest is to reduce your electronic device use. You could also consider EMF block paint to prevent EMF radiation from reaching your home. Another method to protect your house from EMF radiation would be to put up a RF shielding canopy. It is a type made of net which contains EMF shielding. It is utilized to prevent EMFs from entering rooms. Another option is to have your house equipped with a conductive enclosure. These enclosures are known as Faraday cages.
Numerous studies have demonstrated studies have shown that nonionizing EMF can cause antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed to be based on the downregulation of cancer stem cells. This could be the reason for the long-term effects observed in some patients with advanced HCC. However, the mechanism of AM EMF's effects on patients with cancer is not yet clear.
The effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo was studied in mice. The tumours were divided in three different groups. One group did not have exposure to RF EMF. Another group of participants was subjected RF EMF at a frequency that is similar to that used in humans. The third group was exposed to RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on the tumours was assessed against the effect of RCF. The results indicated that tumours treated with HCCMF had significant shrinkage. However, the tumors treated with RCF showed no evidence of shrinkage of the tumor.
The mechanism behind cancer-specific AM RF EMF could be based on the fact that tumor cells require Cav3*2 voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is mediated by CACNA1H which is a protein that mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H may have broader implications for treatment and diagnosis of different cancers.
The tumours in those in the group that were unaffected to EMF from radiofrequency, and fed a standard mouse diet. The tumours in those in the HCCMF group were treated with Huh7 cells at the time they were 5 to 7 weeks old. The tumors were then killed in cases of excessive burden.
The tumours from the three groups also displayed different growth curves. The HCCMF-treated tumors showed a significant decrease in tumour size after eight weeks. However, tumors that were treated using RCF did not show any shrinkage. The difference was substantial. The tumors treated by RCF were able to show necrosis, which is common when tumors are that are exposed to RCF. The possibility is that the necrosis is caused by the lack of oxygen in larger tumors.
In sum, the results show an AM-RF EMF is a powerful source of anticancer effects in vitro and in vivo. Helpful hints of studies have proven that AM RF EMF produces measurable reduction in tumours in HCC patients. emf blockers is possible that AM RF EMF produces these effects because of CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained impact on the growth of HCC tumors in living tissue.
