Whether you live in an apartment or house or just would like to keep your home free of EMFs There are a variety options to limit your exposure. One of the most effective is to limit the use of electronic devices. You could also consider EMF blocker paint to block EMF radiation from reaching your home. Another easy way to protect your home from EMF radiations is to put up a RF shielding canopy. It's a kind of net that has EMF shielding. It's used to stop EMFs from entering rooms. Another alternative is to have your house equipped with an enclosure that is conductive. These enclosures are known as Faraday cages.
Numerous studies have demonstrated studies have shown that the nonionizing EMF can cause antiproliferative effects in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed result from the deregulation in cancer-related stem cells. This could explain the long-term responses seen in some patients with advanced HCC. However, the reason behind AM RF EMF's effect in patients with cancer is not evident.
Aspects from AM RF EMFs on HCC tumour growth in vivo were examined in mice. The tumors were separated into 3 groups. One group did not have exposure to RF EMF. Second group members were exposed RF EMF at the same frequency to the frequency used by humans. Third group members were exposed to the RF EMF in HCC-specific frequencies. emf blocking of HCCMF on tumours was evaluated against that of RCF. The results showed that the cancers treated with HCCMF showed significant shrinkage. However, the tumours treated with RCF showed no evidence of shrinkage in the tumour.
The mechanism of tumour-specific AM RF EMF may be driven by the fact that tumor cells require Cav3*2 voltage calcium channels to promote proliferation and down-regulation. block emf upon HCC cells is controlled by CACNA1H the protein which is responsible for the influx of Ca2+ specific to tumours. The results suggest that CACNA1H may have broader implications for diagnosis and treatment of a variety of cancers.
The tumours of those in the group that were unaffected EMF from RF, and fed a normal mouse diet. The tumours in those in the HCCMF group were treated with Huh7 cells after they were five-seven weeks old. The tumours were then euthanized after they had a high burden.
emf blocking in the three groups also showed distinct growth curves. The tumours treated with HCCMF showed a significant decrease in size of the tumor after 8 weeks. However, the tumours treated with RCF did not show any signs of shrinkage. The difference was highly significant. The tumours treated with RCF were able to show necrosis, which is common in tumours that are exposed to RCF. The possibility is that the necrosis is caused by a lack of oxygen in the larger tumours.
In sum, the results suggest an AM-RF EMF exhibits anticancer effects in vitro and in vivo. Several studies have shown it is true that AM RF EMF produces measurable reduction in tumours within HCC patients. The possibility is that the AM EMF causes these effects due to CACNA1H, a protein that is involved in the tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting influence on the development of HCC tumors in living tissue.