https://yarnself9.bravejournal.net/post/2023/04/08/EMF-Block-Paint-plus-EMF-Shielding5 if you live in a house or apartment, or you just want to keep your home free of electromagnetic fields, there are a number of methods to reduce exposure. emf blocking of the most effective is to restrict the use of electronic devices. You can also turn to EMF block paint to prevent EMF radiation from reaching your house. Another easy way to protect your house from EMF radiation is to install a RF shielding canopy. This is a cloth made of net which contains EMF shielding. It is utilized to block EMFs from entering a room. Another alternative is to have your home equipped with an enclosure that is conductive. They are referred to as Faraday cages.
Numerous studies have demonstrated studies have shown that the nonionizing EMF has antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed be based on the downregulation the cancer stem cell. This could explain the long-term response seen in patients with advanced HCC. But, the reason for AM EMF's effects on cancer patients isn't clear.
Effects on the effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo was studied in mice. The tumors were separated into three groups. The first group was not exposed to RF EMF. Another group of participants was subjected RF EMF at the same frequency to that of humans. Third group members were exposed to RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on the tumours was compared to that of RCF. The results showed that the cancers treated with HCCMF showed significant shrinkage. However, tumours treated with RCF showed no evidence of shrinkage in the tumour.
The mechanism of cancer-specific AM RF EMF could be based on the fact that tumor cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is controlled by CACNA1H the protein which mediates tumour-specific Ca2+ influx. The findings suggest that CACNA1H may have broader implications for the diagnosis and treatment of a variety of cancers.
The tumors in the controls were never exposed RF EMF, and were fed a standard mouse diet. The tumors of those in the HCCMF group were treated with Huh7 cells when they were 5 to 7 weeks old. emf blockers were removed after they had a high burden.
The tumors of the three groups also showed different growth curves. The tumours treated with HCCMF saw a significant decrease in the size of the tumour after 8 weeks. However, the tumors which were treated by RCF showed no shrinkage. The difference was significant. The tumours treated with RCF had necrosis that is common when tumors are exposed to RCF. The possibility is that the necrosis was caused by the lack of oxygen in larger tumors.
In sum, the results indicate the fact that AM EMF has anti-cancer properties in vitro as well as in vivo. Several studies have shown it is true that AM RF EMF produces measurable reduction in tumours within HCC patients. There is a possibility that AM RF EMF causes these effects due to CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting influence on the development of HCC tumours in living tissue.