If you reside in an apartment or house, or you just want to keep your home free of electromagnetic fields There are a variety of ways you can reduce exposure. One of the most effective is to restrict the usage of your electronic devices. You could also consider EMF blocker paint to block EMF radiation from entering your house. Another easy way to protect your home against EMF radiation is to install an RF shielding canopy. emf blocker is a type of net that has EMF shielding. It's used to stop EMFs from entering rooms. Another alternative is to have your home equipped with a conductive enclosure. emf blocking are known as Faraday cages.
A number of studies have proven how the EMF that is not ionized can cause anti-proliferative effects on HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed result from the deregulation the cancer stem cell. This could be the reason for the long-term effects observed in some patients with advanced HCC. But, the reason for AM EMF's effects on cancer patients is not yet clear.
Effects on the effects of AM RF EMFs on HCC tumour growth in vivo were examined in mice. The tumours were divided in three different groups. First, the group that was unaffected to RF EMF. The second group was exposed RF EMF at a frequency that is similar to the frequency used by humans. The third group was exposed RF EMF with HCC-specific modulation frequencies. The effects of HCCMF on tumours was compared to that of RCF. The results revealed that tumors treated by HCCMF showed significant shrinkage. However, the tumours treated with RCF did not show any evidence of shrinkage in the tumour.
The reason for tumor-specific AM RF EMF might be driven by the fact that tumour cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effects upon HCC cells is controlled by CACNA1H which is a protein which regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H may have broader implications for treatment and diagnosis of various cancers.
The tumors in the control group were not exposed EMF from RF, and fed a normal mouse diet. The tumors of the HCCMF group were treated with Huh7 cells after they were five-seven weeks old. The tumors were then killed when they showed excessive burden.
The tumors of the three groups also showed different growth curves. The tumours treated with HCCMF had a significant reduction in the size of the tumour after eight weeks. However, the tumours that were treated using RCF showed no reduction in size. The difference was highly significant. The tumors treated by RCF were able to show necrosis that is common in tumors that have been that are exposed to RCF. There is a possibility that the necrosis was due to a lack of oxygen in the more invasive cancers.
In conclusion, the findings indicate the fact that AM EMF is a powerful source of anticancer activity in vitro and in the vivo. emf blocking of studies have proven the fact that AM RF EMF produces measurable tumour shrinkage for HCC patients. The possibility is that the AM EMF produces these effects through CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting effect on the development of HCC tumors in living tissue.