No matter if you live in an apartment or a house or just would like to keep your home free from EMFs There are a variety options to limit your exposure. One of the most straightforward is to restrict the use of electronic devices. It is also possible to use EMF blocker paint to block EMF radiation from entering your home. Another method to protect your house against EMF radiation is to install an RF shielding canopy. It is a type of net that contains EMF shielding. It's used to block EMFs from entering rooms. Another alternative is to have your house equipped with a conductive enclosure. They are referred to as Faraday cages.
Several studies have shown that the non-ionizing RF EMF can cause antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to result from the deregulation in cancer-related stem cells. This could explain the long-term responses seen in certain patients suffering from advanced HCC. But, the reason for AM EMF's effects on patients with cancer is not yet clear.
Aspects on the effects of AM RF EMFs on HCC tumour growth in vivo were studied in mice. The tumors were divided in three different groups. The first group was not exposed to RF EMF. Second group members were exposed to RF EMF at frequencies similar to that of humans. emf blocking was exposed to the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumours was evaluated against that of RCF. The results indicated that tumors treated by HCCMF were significantly shrinking. However, the tumors treated with RCF didn't show evidence of tumour shrinkage.
The mechanism behind tumour-specific AM RF EMF could be based on the fact that tumor cells require Cav3*2 T-type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is mediated through CACNA1H the protein that regulates the Ca2+ influx specific to tumors. The findings suggest that CACNA1H could have more broader implications for the diagnosis and treatment of a variety of cancers.
The tumours in the control group were not exposed EMF from radiofrequency, and fed a normal mouse diet. The tumours in the HCCMF group were infected with Huh7 cells after they were between five and seven weeks old. The tumors were then killed when they showed excessive burden.
The tumors in the three groups also showed different growth curves. The tumors treated with HCCMF showed a significant decrease in tumour size after eight weeks. However, block emf treated with RCF showed no signs of shrinkage. The difference was highly significant. emf blockers treated with RCF showed necrosis, which is common in tumours exposed to RCF. It is possible that the necrosis was due to the lack of oxygen in larger cancers.
In conclusion, the findings show an AM-RF EMF is a powerful source of anti-cancer properties in vitro as well as in vivo. Numerous studies have demonstrated that AM RF EMF produces measurable reduction in tumours within HCC patients. The possibility is that the AM EMF produces these effects due to CACNA1H, a protein involved in the tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained influence on the growth of HCC tumours in living tissue.