EMF Block Paint and EMF Shielding

· 2 min read
EMF Block Paint and EMF Shielding

No matter if you live in a house or apartment, or you just would like to keep your home free of EMFs There are plenty of methods to reduce exposure. One of the easiest is to restrict the use of electronic devices. You can also turn to EMF block paint to stop EMF radiation from reaching your house. Another easy way to protect your home against EMF radiation is to use an RF shielding cover. It is a type made of net which contains EMF shielding. It's used to prevent EMFs from entering a room. Another option is to get your home equipped with an electrical enclosure. They are referred to as Faraday cages.

Numerous studies have demonstrated studies have shown that the nonionizing EMF can cause antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to result from the deregulation in cancer-related stem cells. This could be the reason for the long-term effects observed in certain patients suffering from advanced HCC. However, the reason behind AM EMF's effects on cancer patients isn't yet clear.

Aspects from AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were examined in mice. The tumors were separated into 3 groups. The first group was not exposed RF EMF. Second group members were exposed RF EMF at frequencies similar to the frequency used by humans. Third group members were exposed to RF EMF at HCC-specific modulation frequencies. The impact of HCCMF on tumours was evaluated against that of RCF. The results revealed that tumours treated with HCCMF were significantly shrinking. However, tumours treated with RCF showed no evidence of shrinkage of the tumor.

The mechanism behind tumor-specific AM RF EMF may be based on the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is mediated through CACNA1H which is a protein which is responsible for the influx of Ca2+ specific to tumours. The results suggest that CACNA1H could have more broader implications for the diagnosis and treatment of a variety of cancers.

emf blocker  of the control group were not exposed to EMF from radiofrequency, and fed a standard mouse diet. The tumours in those in the HCCMF group were infected with Huh7 cells after they were five-seven weeks old. The tumors were then killed when they showed excessive burden.

The tumors of the three groups showed different growth curves. The tumors treated with HCCMF had a significant reduction in the size of the tumour after eight weeks. However, the tumors which were treated by RCF showed no reduction in size. The difference was significant. The tumors treated by RCF showed necrosis, which is common in tumours that are exposed to RCF.  emf blocker  is possible that this necrosis was caused by a lack of oxygen in the more invasive cancers.


In summary, the results indicate the fact that AM EMF is a powerful source of anticancer activity in vitro and in vivo. Numerous studies have demonstrated that AM RF EMF produces measurable reduction in tumours in HCC patients. The possibility is that the AM EMF triggers these effects through CACNA1H which is a protein involved in the tissue-specific Ca2+ influx. In  emf blocking , AM RF EMF may cause a lasting impact on the development of HCC tumours in the vivo.